The PS5 dev kit looks hugely different from Sony’s final console
Its casing is radically different from the PS5’s retail version, and it’s outfitted with several more ports and inputs. The unit featured in the YouTuber’s video hasn’t been activated, however, so can’t boot up any software past the kit’s home screen.The V-shaped device features an integrated LCD display on its faceplate, a pair of heavy-duty exhaust ports running down either side, as well as grooves that let multiple kits stack on top of one another.
The guts of the machine are different too. It contains nearly 2.5TB of memory – far greater than the 825GB SSD included in the retail PS5 – as well as the capacity for expanded storage. Unlike the consumer console, it doesn’t include a disc drive.
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The architecture of clonal expansions in morphologically normal tissue from cancerous and non-cancerous prostates - Molecular CancerBackground Up to 80% of cases of prostate cancer present with multifocal independent tumour lesions leading to the concept of a field effect present in the normal prostate predisposing to cancer development. In the present study we applied Whole Genome DNA Sequencing (WGS) to a group of morphologically normal tissue (n = 51), including benign prostatic hyperplasia (BPH) and non-BPH samples, from men with and men without prostate cancer. We assess whether the observed genetic changes in morphologically normal tissue are linked to the development of cancer in the prostate. Results Single nucleotide variants (P = 7.0 × 10–03, Wilcoxon rank sum test) and small insertions and deletions (indels, P = 8.7 × 10–06) were significantly higher in morphologically normal samples, including BPH, from men with prostate cancer compared to those without. The presence of subclonal expansions under selective pressure, supported by a high level of mutations, were significantly associated with samples from men with prostate cancer (P = 0.035, Fisher exact test). The clonal cell fraction of normal clones was always higher than the proportion of the prostate estimated as epithelial (P = 5.94 × 10–05, paired Wilcoxon signed rank test) which, along with analysis of primary fibroblasts prepared from BPH specimens, suggests a stromal origin. Constructed phylogenies revealed lineages associated with benign tissue that were completely distinct from adjacent tumour clones, but a common lineage between BPH and non-BPH morphologically normal tissues was often observed. Compared to tumours, normal samples have significantly less single nucleotide variants (P = 3.72 × 10–09, paired Wilcoxon signed rank test), have very few rearrangements and a complete lack of copy number alterations. Conclusions Cells within regions of morphologically normal tissue (both BPH and non-BPH) can expand under selective pressure by mechanisms that are distinct from those occurring in adjacent cancer, but that are allied t
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