Scientists at The Wistar Institute have discovered a potential target for gastric cancers associated with Epstein-Barr Virus, and their study results are published in the journal mBio.
plays a significant role in regulating the EBV latency-lysis cycle within the cancer cells.
To disrupt this epigenetic profile, the researchers turned to decitabine, a compound known for its ability to reduce DNA methylation levels . Tempera and his co-authors treated twothat were derived from EBVaGC tumors with decitabine. The cell lines that received the treatment demonstrated massive reductions in DNA methylation across the genome relative to the control as assessed by a variety of epigenetic assay techniques.
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