Inflammation is a standard part of our bodies' immune system response. But sometimes this response becomes hyperactivated in our lungs, causing inflammation to continue unchecked, which can be fatal. Many deaths from COVID-19 have been due to excessive inflammation, which results in acute lung injury.
A group of researchers at the University of Illinois Chicago have investigated how lungs counterbalance inflammation. Their work points to cells in the lung that reduce inflammation by removing cellular debris, ingesting harmful bacteria and releasing anti-inflammatory proteins. What's more, these cells can be trained by an initial infection to do this job even better during a subsequent infection.
The scientists demonstrated that injecting these trained cells into mice helped keep them alive after an infection with pneumonia. These results suggest that such trained cells could become part of a cell therapy that prevents excessive inflammation. The research is published in thehelped reduce the severity of inflammation brought on by a second exposure to the bacterial toxin one week later.
The cells appear to become particularly good at removing post-infection debris—such as cellular debris from
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Lung cells can be trained to counteract inflammationInflammation is a standard part of our bodies' immune system response. But sometimes this response becomes hyperactivated in our lungs, causing inflammation to continue unchecked, which can be fatal. Many deaths from COVID-19 have been due to excessive inflammation, which results in acute lung injury.
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